It demonstrates how powerful these molecules are to damaging and killing the cell. Such medications might re-enter the pipeline, if this free-radical creating pathway is exploited to lower the therapeutic dose, producing dangerous drugs safer formerly.. A chink in bacteria’s armor Biomedical researchers at Boston University’s College of Engineering may have discovered the path toward growing better drugs capable of defeating so-called superbugs, bacteria which have designed resistance to common antibiotics.Related StoriesAir Liquide Healthcare launches a website dedicated to rest apneaWhy do we sleep?S. Product sales of approximately $437 million, according to IMS Health data.
Francisco M. Marty, M.D., Drew J. Winston, M.D., Scott D. Rowley, M.D., Estil Vance, M.D., Genovefa A. Papanicolaou, M.D., Kathleen M. Mullane, D.O., Thomas M. Brundage, M.S., Alice T. Robertson, Ph.D., Susan Godkin, R.Ph.D., and Michael Boeckh, M.D. For the CMX001-201 Clinical Study Group: CMX001 to Prevent Cytomegalovirus Disease in Hematopoietic-Cell Transplantation Cytomegalovirus contamination is a common cause of illness after allogeneic hematopoietic-cell transplantation.1,2 CMV seropositivity in transplant recipients is connected with an increased threat of death after transplantation also, despite prophylactic and preemptive strategies with available antiviral agents.3-5 Although valganciclovir is approved for prophylaxis against CMV infection after solid-organ transplantation, its use is limited by myelosuppression, after hematopoietic-cell transplantation particularly.3,6-8 Thus, there is an unmet dependence on effective medications against CMV infection that have an improved safety profile.